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In both cell lines, a constant observation was the increased or maintained high levels of glutamine and glucose (Figs

doi: 10.3389/fphar.2021.627533

Dan and Baldwin [81] revealed that insulin and TNF activated mTOR through both the AKT/mTOR and the AKT/IKK/mTOR pathways, whereas TNF also activated mTOR via the IKK-mediated signaling independently of the PI3K/AKT pathway in several cell types

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A., Herring, A