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By replenishing the intracellular pools of this master antioxidant, the body can begin to shield its vulnerable mitochondria from ongoing free radical damage

doi: 10.1007/BF00399926

In the long-term studies conducted by Khavinson and his team, subjects followed for up to 15 years showed no significant toxicity or negative outcomes attributable to epithalon use

Alcohol Clin Exp Res, 2011

Therefore, most data are rather descriptive based on knockout, inhibition, or overexpression of redoxins and subsequent measurement of lipid peroxidation and/or rescue by ferroptosis inhibitors/inducers

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