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Specifically, hepatoma cells with low expression of GOT2 showed a high dependence on Gln metabolism by increasing Gln metabolism, nucleotide synthesis, and glutathione synthesis to support cellular antioxidants (Fig

In an in vitro model (human mesangial cells stimulated by Gd-IgA1), cell viability was markedly reduced, accompanied by lipid peroxidation and characteristic mitochondrial changes associated with ferroptosis

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