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Mechanistically, GSH depletion does not cause global protein oxidation but instead selectively oxidizes and destabilizes fatty acid synthase while decreasing lipogenic gene expression at the transcriptional level, collectively suppressing lipogenesis
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GSH depletion triggers cell cycle arrest in G1/S checkpoint and induces SIPS SIPS is well-characterized in cells treated with exogenous oxidants such as tBH and H 2 O 2 10,11
We also reported that aged skeletal muscles display a progressive decline of adipose triglyceride lipase (ATGL), which could be responsible for increased intramyocellular lipid droplets [101]
