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TLR1, TLR2 and TLR4 activation results in augmented mitochondrial ROS production following recruitment of mitochondria to macrophage phagosomes, leading the way to increased mitochondrial and cellular ROS generation (West et al., 2011)

In contrast, our results demonstrated that the Q 67 residue is critical for GSH binding

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Systems toxicology: modelling biomarkers of glutathione homeostasis and paracetamol metabolism

In 970 L of buffer (100 mM Tris-HCl, 1 mM EDTA, pH 8.2), 10 L of homogenates and 20 L pyrogallol 13 mM were mixed

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